Tuesday, December 02, 2003
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Neurology for MRCPsych
1. Ankle & knee jerk are both characteristically lost in infectious polymyalgia F
2. Ankle & knee jerk are both characteristically lost in Motor neurone disease F
3. Ankle & knee jerk are both characteristically lost in Tabes dorsalis F
4. Ankle & knee jerk are both characteristically lost in Fredrick’s ataxia T
5. Ankle & knee jerk are both characteristically lost in Cervical spine lesions F
6. Posterior column lesion results in the loss of pain sensation F
7. Posterior column lesion results in the loss of light touch T
8. Posterior column lesion results in the loss of vibration sense T
9. Posterior column lesion results in the loss of proprioception T
10. Posterior column lesion results in the loss of discriminatory touch T
11. Closure of the eyes attenuates alpha rhythm on EEG
12. EEG tracing is abolished in pre hepatic coma
13. EEG tracking is maximal in the occipital lobes
14. EEG amplitude can be asymmetrical normally
15. EEG Alpha rhythms are continual through out sleep
16. The optic reflex depends on Optic radiation
17. The optic reflex depends on Edinger-westphal nucleus T
18. The optic reflex depends on occipital cortex
19. The optic reflex depends on Occulomotor nerve T
20. The optic reflex depends on Ciliary muscles T
21. CJD is rare under the age of 40 T
22. CJD is commoner in rural areas F
23. CJD shows characteristic EEG changes (this is true for sporadic CJD)
24. CJD has only been transmitted to experimental animals via brain tissue from an infected object F
25. CJD is one of the spongiform encephalopathies T
26. Features of pseudobulbar palsy include brisk jaw jerk T
27. Features of pseudobulbar palsy include emotional lability T
28. Features of pseudobulbar palsy include fasciculation of the tongue F
29. Features of pseudobulbar palsy include absent gag reflex F
30. Features of pseudobulbar palsy include Dysphagia T
31. CT scan of the head is the initial investigation of in subarachnoid haemorrhage T
32. CT scan of the head demonstrates Nigro striatal degeneration in Parkinson’s disease F
33. CT scan of the head is often normal within 6 hours of a large infarct T
34. Fresh blood shows up as dark areas on CT scan of the head F
35. CT scan of the head demonstrates all space occupying lesions
36. MRI of the head is not very sensitive for detecting whiter matter changes in multiple sclerosis F
37. Claustrophobia is a relative contraindication for MRI of the head T
38. A pacemaker is an absolute contra indication for MRI of the head T
39. MRI of the head demonstrate classification better than CT scan F
40. MRI of the head is useful in temporal lobe epilepsy F
41. Lumber puncture is always contra indicated if papilloedema is present F
42. Lumber puncture is safe if a frontal lobe mass is present F
43. Lumber puncture is safe in benign intra cranial hypertension T
44. Lumber puncture can be diagnostic in SAH T
45. Micropsia may occur in aura with temporal lobe focus T
46. pareidolia may occur in aura with temporal lobe focus F
47. Tinnitus may occur in aura with temporal lobe focus T
48. A smell of burning rubber may occur in aura with temporal lobe focus
49. Xanthopia may occur in aura with temporal lobe focus T
50. Forced thinking may occur in aura with temporal lobe focus T
51. Depersonalisation may occur in aura with temporal lobe focus T
52. The punch drunk syndrome is associated with cerebral atrophy T
53. The punch drunk syndrome is associated with aphasia F
54. The punch drunk syndrome is associated with ataxia
55. The punch drunk syndrome is associated with Personality changes T
56. The punch drunk syndrome is associated with mask faces T
57. Fasciculation of the upper limb Is commonly associated with MND T
58. Dysphagia is commonly associated with MND T
59. Dysarthria is commonly associated with MND T
60. Amnesia is commonly associated with MND F
61. Uncontrolled weeping is commonly associated with MND T
62. Neuro fibrillary tangles are seen in Alzheimer’s disease T
63. Neuro fibrillary tangles are seen in Down’s syndrome T
64. Neuro fibrillary tangles are seen in Pick’s disease F
65. Neuro fibrillary tangles are seen in Lewy body dementia T
66. Neuro fibrillary tangles are seen in Punch drunk syndrome T
67. Parietal lobe lesions can produce superior quadrant hemianopia F
68. Dominant parietal lobe lesions can produce left-right disorientation T
69. Parietal lobe lesions can produce hemi-somatoagnosia T
70. Parietal lobe lesions can produce alexia with agraphia T
71. Non-dominant parietal lobe lesions can produce Sensory dissociation F
72. Non-dominant parietal lobe lesions can lead finger agnosia F
73. Dominant parietal lobe lesions can lead to agraphia T
74. Posterior parietal lobe damage can lead to fluent aphasia. T
75. GCS is a good predictor of long term outcome in severe head injury F
76. Post traumatic epilepsy is more common after a close head injury F
77. The length of post traumatic amnesia correlates with cognitive impairment T
78. Blood is always found in the CSF in severe head injuries F
79. in severe head injuries the brain is more seriously injured when it is in a fixed position F
80. Clouding of consciousness is seen in Korsakoff’s psychosis F
81. Clouding of consciousness is seen in Wernicke’s encephalopathy T
82. Clouding of consciousness is seen in Alcohol hallucinosis F
83. Clouding of consciousness is seen in Gertsman syndrome F
84. Clouding of consciousness is seen in Ganser syndrome T
85. In normal EEG alpha waves are attenuated when eyes are open T
86. In normal EEG alpha waves can have different frequency in 50%of patients T
87. In normal EEG alpha waves may disappear in hepatic coma T
88. In normal EEG alpha waves are seen predominantly in the occipital region T
89. In normal EEG alpha waves are increased by lithium F
90. The caudate is part of limbic system
91. The caudate is altered in Parkinson disease
92. The caudate is connected to substantia nigra
93. The caudate is part of telecephalon
94. The caudate contains glia
95. Oculomotor nerve carry parasympathetic nerve fibres
96. Facial nerve carry parasympathetic nerve fibres
97. Trigeminal nerve carry parasympathetic nerve fibres
98. Vagus nerve carry parasympathetic nerve fibres T
99. Hypoglossal nerve carry parasympathetic nerve fibres F
100. Features of dominant parietal lobe lesions include astreognosis T
101. Features of dominant parietal lobe lesions include dyscalculia T
102. Features of dominant parietal lobe lesions include Sensory inattention T
103. Motor neurone disease may present with Schizophrenoform Psychosis F
104. Motor neurone disease may present with Frontal lobe atrophy or dementia F
105. Motor neurone disease may present with emotional lability T
106. Motor neurone disease may present with spastic paraplegia T
107. More than 2% glucose in CSF is seen in Multiple Sclerosis F
108. More than 2% glucose in CSF Guillane-Barre syndrome T
109. More than 2% glucose in CSF Spinal epidural abscess T
110. More than 2% glucose in CSF Dementia F
111. More than 2% glucose in CSF Acoustic neuroma F
112. Multiple Sclerosis may present with Dysphagia F
113. Multiple Sclerosis may present with Euphoria T
114. Multiple Sclerosis may present with Depression T
115. Multiple Sclerosis may present with Dysarthria T
116. Multiple Sclerosis may present with Cortical blindness
117. Ejaculation is associated with increased parasympathetic activity T
118. Relaxation of the ciliary muscles is associated with increased parasympathetic activity F
119. Relaxation of external anal sphincter is associated with increased parasympathetic activity F
120. Sweating is associated with increased parasympathetic activity T
121. Salivation is associated with increased parasympathetic activity T
122. EEG alpha rhythm decreases to 8Hz after 65 T
123. Temazepam can increase beta fast wave activity on EEG T
124. EEG shows diffuse slow waves even after a month following ECT T
125. Normal EEG makes the diagnosis of Advanced Alzheimer’s disease unlikely T
126. Normal EEG makes the diagnosis of Petit mal epilepsy unlikely F
127. Normal EEG makes the diagnosis of Hypsarrythmia unlikely T
128. Normal EEG makes the diagnosis of sub-acute Sclerosing leuco- encephalitis unlikely T
129. Normal EEG makes the diagnosis of Cerebellar glioma unlikely T
Hypsarrythmia is an age related EEG pattern usually associated with infantile spasm & west syndrome
130. Caudate nucleus is involved in Parkinson’s disease
131. Caudate nucleus together with lenticular nucleus forms the striatum
132. Caudate nucleus projects to sustantia nigra
133. Caudate nucleus is rich in dopamine receptors
134. Mild head injury may cause Intolerance to alcohol F
135. Mild head injury may cause Insomnia T
136. Mild head injury may cause dizziness T
137. Mild head injury may cause Hypersensitivity to sound T
138. Mild head injury may cause Epilepsy F
139. Both Ankle & knee jerks are lost in Fredrick’s ataxia T
140. Both Ankle & knee jerks are lost in Cervical spondylosis F
141. Both Ankle & knee jerks are lost in Polyneuropathy T
142. Both Ankle & knee jerks are lost in Tabes dorsalis F
143. Both Ankle & knee jerks are lost in MND F
144. Eye signs are seen in Tay sachs disease T
145. Eye signs are seen in Phenile keton urea F
146. Eye signs are seen in Wilson’s disease T
147. Eye signs are seen in Wernicke’s encephalopathy T
148. Eye signs are seen in Multiple Sclerosis T
149. Imitative behaviour suggests a frontal lobe lesion F
150. Perseveration suggests a frontal lobe lesion T
151. Having difficulties in card sorting test suggests a frontal lobe lesion T
152. Dyscalculia suggests a frontal lobe lesion
153. Disinhibited behaviour suggests a frontal lobe lesion
154. Sequelae of closed head injury include Insomnia T
155. Sequelae of closed head injury include Epilepsy T
156. Sequelae of closed head injury include Intolerance to Alcohol F
157. Sequelae of closed head injury include Hyper-sensitivity to sound T
158. Sequelae of closed head injury include memory loss T
Retro grade amnesia (time between the injury & last memory of events before injury) is a Poor prognostic indicator.
Anterograde amnesia (time between injury & recovery of normal memory) is a good prognostic indicator.
The longer the amnesia the worse the prognosis. Intra cranial bleeding & penetrating injuries are associated with poor prognosis.
Sequelae of head injury include:
· Neurosis, depression, fatigue, anxiety, irritability, excessive sensitivity to noise & somatic complaints.
· Personality change
· Psychosis
· Cognitive impairment
· Epilepsy especially with penetrating injuries
159. Neurological examination in Parkinson’s disease reveals large sweeping hand writing F
160. Neurological examination in Parkinson’s disease reveals decrease voluntary muscle power F
161. Neurological examination in Parkinson’s disease reveals Increased fatigability F
162. Neurological examination in Parkinson’s disease reveals Bradykinesia T
163. Neurological examination in Parkinson’s disease reveals Failed habituation to glabellar tab T
164. Characteristic features of CJD include association with a transmissible agent T
165. Characteristic features of CJD include Myoclonic jerks T
166. Characteristic features of CJD include Ataxia T
167. Characteristic features of CJD include spontaneous remission F
168. Characteristic features of CJD include akinetic mutism in late stages T
169. Characteristic features of CJD include repetitive sharp wave complexes on EEG T
170. Frontal lobe meningioma may cause calcification on skull X ray T
171. Frontal lobe meningioma may cause epilepsy T
172. Frontal lobe meningioma may cause ataxia T
173. Frontal lobe meningioma may cause personality change T
174. Frontal lobe meningioma is best diagnosed by CT scan F
175. Features of left parietal lobe lesion include apathy & psychomotor retardation F
176. Features of left parietal lobe lesion include alexia & agraphia T
177. Features of left parietal lobe lesion include hemi-somatoagnosia F
178. Features of left parietal lobe lesion include amnesia F
179. Features of left parietal lobe lesion include Euphoria F
180. Fast beta activity on EEG may be caused by Chlorpromazine F
181. Fast beta activity on EEG may be caused by Amitryptiline F
182. Fast beta activity on EEG may be caused by phenobarbiton T
183. Fast beta activity on EEG may be caused by Lithium F
184. Fast beta activity on EEG may be caused by Diazepam T
185. De javu is one of the paramnesias T
186. De javu can be part or a whole of the aura in temporal lobe epilepsy T
187. De javu is a spurious sense of unfamiliarity F
188. De javu is not compelling as a feeling & is never remembered F
189. De javu occurs in normal people T
190. Signs in Huntington’s chorea present characteristically in late teens F
191. Huntington’s chorea is autosomal dominant T
192. Huntington’s chorea may display anticipation through generations T
193. In Huntington’s chorea psychotic features may precede abnormal movements T
194. Atrophy of the caudate nucleus is a typical feature of Huntington’s chorea T
195. Personality disorder is more common in idiopathic than epilepsy of known aetiology F
196. Personality disorder is commoner in grand mal than in petit mal T
197. There is a specific epileptic personality F
198. Hyper sexuality is the commonest sexual disorder in epilepsy F
199. Primary epilepsy account for <50% of all patients F
200. Retrograde amnesia is a valid & valuable guide to the severity of head injury F
201. Pre morbid personality is important in determining ensuing psychiatric disturbance in patient with head injury T
202. Post-traumatic epilepsy develops in 25% of patients following head injury
203. Cognitive impairment is the commonest psychiatric disorder following head injury F
204. The incidence of psychiatric Sequelae is higher in children following head injury F
205. A parietal lobe lesion may cause motor Jacksonian fits F
206. A parietal lobe lesion may cause astereognosis T
207. A parietal lobe lesion may cause grasp reflex F
208. A parietal lobe lesion may cause anosognosia T
209. A parietal lobe lesion may cause homonymous hemianopia T
210. Frontal lobe syndrome is frequently associated with hyper-sexuality F
211. Frontal lobe syndrome is frequently associated with fatuous mood T
212. Frontal lobe syndrome is frequently associated with disinhibition T
213. Frontal lobe syndrome is frequently associated with behavioural changes in the absence of neurological signs T
214. Frontal lobe syndrome is frequently associated with marked loss of memory F
215. Multiple sclerosis is associated with Optic neuritis T
216. Multiple sclerosis is associated with Euphoria T
217. Multiple sclerosis is associated with nystagmus T
218. Abnormalities of gait are commonly found in normal pressure hydrocephalus T
219. Abnormalities of gait are commonly found in Folate deficiency F
220. Abnormalities of gait are commonly found in Fredrick’s ataxia T
221. Abnormalities of gait are commonly found in Sturge weber syndrome F
222. Abnormalities of gait are commonly found in Parkinson’s disease T
223. Cerebellar lesions may cause Hypotonicity T
224. Cerebellar lesions may cause reduced deep tendon reflexes T
225. Cerebellar lesions may cause tremor at rest F
226. Cerebellar lesions may cause Dysarthria T
227. Cerebellar lesions may cause sphincter disturbance F
228. Loss of sensation is seen immediately following acute transection of the spinal cord T
229. Loss of muscle power is seen immediately following acute transection of the spinal cord T
230. Spasticity is seen immediately following acute transection of the spinal cord F
231. Loss of reflexes is seen immediately following acute transection of the spinal cord T
232. Vasodilatation is seen immediately following acute transection of the spinal cord T
233. Frontal lobe lesions are associated with inability to plan actions T
234. Frontal lobe lesions are associated with apathy T
235. Frontal lobe lesions are associated with Positive grasp reflex T
236. Frontal lobe lesions are associated with incontinence T
237. Frontal lobe lesions are associated with aphasia T
238. Frontal lobe lesions are associated with contra-lateral upper limb weakness T
239. Frontal lobe lesions are associated with topographical disorientation F
240. Parietal lobe lesions are associated with topographical disorientation T
241. Parietal lobe lesions are associated with Visual agnosia
242. Parietal lobe lesions are associated with Sensory inattention T
243. Parietal lobe lesions are associated with Urinary incontinence F
244. Parietal lobe lesions are associated with De Javu phenomenon F
245. Asymmetrical neurological signs are characteristic of idiopathic Parkinson’s F
246. Action tremor is characteristic of idiopathic Parkinson’s F
247. Spastic rigidity is characteristic of idiopathic Parkinson’s F
248. Brady kinesia is characteristic of idiopathic Parkinson’s T
249. Difficulty turning in bed is characteristic of idiopathic Parkinson’s T
250. Paralysis of the right leg is seen after anterior coronary artery occlusion T
251. Paralysis of the left arm is seen after anterior coronary artery occlusion F
252. Grasp reflex is seen after anterior coronary artery occlusion T
253. Right homonymous hemianopia is seen after anterior coronary artery occlusion F
254. Bruca’s aphasia is seen after anterior coronary artery occlusion F
255. Communicating hydrocephalus can cause raised intra-cranial pressure F
256. Sub-arachnoid haemorrhage can cause raised intra-cranial pressure T
257. Sagital vein thrombosis can cause raised intra-cranial pressure T
258. Sub acute sclerosing panenceohalitis can cause raised intra-cranial pressure F
259. Hypertensive encephalopathy can cause raised intra-cranial pressure T
260. Echopraxia is a feature of frontal lobe syndrome F
261. Characteristic features of non dominant parietal lobe lesion include inability to appreciate music F
262. Characteristic features of non dominant parietal lobe lesion inability to plan ahead F
263. Characteristic features of non dominant parietal lobe lesion inability to draw from memory T
264. Characteristic features of non dominant parietal lobe lesion inability to shift from one concept to another F
265. Characteristic features of non dominant parietal lobe lesion inability to learn new words F
266. Characteristic features of communicating hydrocephalus include onset in the second decade of life
267. Characteristic features of communicating hydrocephalus include Osteoporotic changes detected on skull X ray
268. Characteristic features of communicating hydrocephalus include disturbance of gait T
269. Characteristic features of communicating hydrocephalus include dementia T
270. Characteristic features of communicating hydrocephalus include pathognomonic changes on EEG
271. Posterior column of the spinal cord carry sensory impulses concerned with Pain F
272. Posterior column of the spinal cord carry sensory impulses concerned with proprioception T
273. Posterior column of the spinal cord carry sensory impulses concerned with light touch T
274. Posterior column of the spinal cord carry sensory impulses concerned with hot & cold sensation F
275. Posterior column of the spinal cord carry sensory impulses concerned with Vibration sensation T
276. Nystagmus is a recognised feature of Vertibro-basillar artery insufficiency T
277. Nystagmus is a recognised feature of multiple sclerosis T
278. Nystagmus is a recognised feature of Horner’s syndrome F
279. Nystagmus is a recognised feature of barbiturate abuse T
280. Nystagmus is a recognised feature of cerebello-pontine angle tumours T
281. Occlusion of middle cerebral artery leads to partial epsilateral facial weakness F
282. Occlusion of middle cerebral artery leads to contra-lateral exaggerated deep reflexes T
283. Occlusion of middle cerebral artery leads to epsilateral spastic paralysis F
284. Occlusion of middle cerebral artery leads to absence of plantar reflexes F
285. Occlusion of middle cerebral artery leads to difficulties in understanding both spoken & written words T
286. Amnestic syndrome may be caused by bilateral hypocampal infarction T
287. Amnestic syndrome may be caused by Epilepsy T
288. Amnestic syndrome may be caused by Herpes simplex encephalitis T
289. Amnestic syndrome may be caused by Infarction of the sub-thalamic body F
290. Amnestic syndrome may be caused by Closed head injury T
291. Amnestic syndrome may be caused by chronic alcohol abuse T
292. Constructional apraxia frequently appear with disorders of spatial perception T
293. In head injuries post traumatic amnesia can occur without retrograde amnesia T
294. TLE is associated with hypo-sexuality T
295. The juvenile onset Huntington’s chorea has a higher incidence of psychosis than adult onset type T
296. Slowing of speech is an early sign of AIDS dementia T
297. Inappropriate tearfulness is a feature of multiple sclerosis T
298. The duration of retrograde amnesia is useful in determining prognosis after head injury F
299. CT scan is superior to MRI In detecting calcified brain lesions T
300. Automatism is usually seen in clear consciousness F
301. Athetotic movements disappear in sleep T
302. Tinnitus is a characteristic feature of TLE F
303. Jamais vu is a characteristic feature of TLE T
304. Epigastric distress is a characteristic feature of TLE T
305. Olfactory hallucination is a characteristic feature of TLE T
306. Errors of judgement differentiate FL syndrome from mania F
307. Urinary incontinence is a characteristic feature of TLE F
308. Speech centres are normally located in the left cerebral hemispheres T
309. Speech production of the average 2-year-old is known as telegraphic speech T
310. Lumber puncture should be avoided in patients suspected clinically of having cerebral abscess T
311. In Huntington’s chorea rhythmical high amplitude delta activity on the EEG is characteristic F
312. Lesions of the ventro-medial hypothalamus cause increased appetite T
313. Men are more commonly affected by Huntington disease than women F
314. Cognitive impairment occurs early in the course of Huntington disease F
315. Violence is rare during automatism T
316. In De javu the person has feeling of familiarity for events experienced for the first time T
317. In jamais vu an experience that a person has experienced before is not associated with the appropriate feeling of familiarity T
318. The most common psychiatric disorder seen in MS is euphoria F
319. Logoclonia may occur in Parkinson’s T
320. Naming & reading are rarely impaired in pure word deafness F
321. Speech is fluent in jargon Dysphasia T
322. In alexia with agraphia the patient is unable to read, write, speak or understand speech F
323. In Huntington disease the insight is lost early in the course of illness T
324. Tics occur at an equal rate in males & females F F>M
325. Behaviour during automatism is usually inappropriate & purposeless F
326. Olfactory hallucination in epilepsy strongly suggest the presence of a brain tumour F
327. The right side of the brain is the dominant side in most left handed people F
328. In pure word dumbness writing is preserved but speech can not be produced at will T
329. Lesions in the bruca’s area produce expressive dysphasia T
330. Lesion of the dominant parietal lobe causes visuo-spatial problems T
331. Post stroke depression is more common following left sided stroke
332. About 50% of people with learning disabilities suffer from epilepsy
333. Bulbar & pseudo-bulbar palsy can result in logoclonia F
334. In conduction dysphasia the patient can not speak & write but can repeat whatever is spoke to him F
335. In pure agraphia speech is normal T
336. Hemisomato-agnosia is the neglect of one side of the body T
337. Somatop-agnosia is an inability to recognise a neurological deficit occurring in one self F
Neurology for MRCPsych
1. Ankle & knee jerk are both characteristically lost in infectious polymyalgia F
2. Ankle & knee jerk are both characteristically lost in Motor neurone disease F
3. Ankle & knee jerk are both characteristically lost in Tabes dorsalis F
4. Ankle & knee jerk are both characteristically lost in Fredrick’s ataxia T
5. Ankle & knee jerk are both characteristically lost in Cervical spine lesions F
6. Posterior column lesion results in the loss of pain sensation F
7. Posterior column lesion results in the loss of light touch T
8. Posterior column lesion results in the loss of vibration sense T
9. Posterior column lesion results in the loss of proprioception T
10. Posterior column lesion results in the loss of discriminatory touch T
11. Closure of the eyes attenuates alpha rhythm on EEG
12. EEG tracing is abolished in pre hepatic coma
13. EEG tracking is maximal in the occipital lobes
14. EEG amplitude can be asymmetrical normally
15. EEG Alpha rhythms are continual through out sleep
16. The optic reflex depends on Optic radiation
17. The optic reflex depends on Edinger-westphal nucleus T
18. The optic reflex depends on occipital cortex
19. The optic reflex depends on Occulomotor nerve T
20. The optic reflex depends on Ciliary muscles T
21. CJD is rare under the age of 40 T
22. CJD is commoner in rural areas F
23. CJD shows characteristic EEG changes (this is true for sporadic CJD)
24. CJD has only been transmitted to experimental animals via brain tissue from an infected object F
25. CJD is one of the spongiform encephalopathies T
26. Features of pseudobulbar palsy include brisk jaw jerk T
27. Features of pseudobulbar palsy include emotional lability T
28. Features of pseudobulbar palsy include fasciculation of the tongue F
29. Features of pseudobulbar palsy include absent gag reflex F
30. Features of pseudobulbar palsy include Dysphagia T
31. CT scan of the head is the initial investigation of in subarachnoid haemorrhage T
32. CT scan of the head demonstrates Nigro striatal degeneration in Parkinson’s disease F
33. CT scan of the head is often normal within 6 hours of a large infarct T
34. Fresh blood shows up as dark areas on CT scan of the head F
35. CT scan of the head demonstrates all space occupying lesions
36. MRI of the head is not very sensitive for detecting whiter matter changes in multiple sclerosis F
37. Claustrophobia is a relative contraindication for MRI of the head T
38. A pacemaker is an absolute contra indication for MRI of the head T
39. MRI of the head demonstrate classification better than CT scan F
40. MRI of the head is useful in temporal lobe epilepsy F
41. Lumber puncture is always contra indicated if papilloedema is present F
42. Lumber puncture is safe if a frontal lobe mass is present F
43. Lumber puncture is safe in benign intra cranial hypertension T
44. Lumber puncture can be diagnostic in SAH T
45. Micropsia may occur in aura with temporal lobe focus T
46. pareidolia may occur in aura with temporal lobe focus F
47. Tinnitus may occur in aura with temporal lobe focus T
48. A smell of burning rubber may occur in aura with temporal lobe focus
49. Xanthopia may occur in aura with temporal lobe focus T
50. Forced thinking may occur in aura with temporal lobe focus T
51. Depersonalisation may occur in aura with temporal lobe focus T
52. The punch drunk syndrome is associated with cerebral atrophy T
53. The punch drunk syndrome is associated with aphasia F
54. The punch drunk syndrome is associated with ataxia
55. The punch drunk syndrome is associated with Personality changes T
56. The punch drunk syndrome is associated with mask faces T
57. Fasciculation of the upper limb Is commonly associated with MND T
58. Dysphagia is commonly associated with MND T
59. Dysarthria is commonly associated with MND T
60. Amnesia is commonly associated with MND F
61. Uncontrolled weeping is commonly associated with MND T
62. Neuro fibrillary tangles are seen in Alzheimer’s disease T
63. Neuro fibrillary tangles are seen in Down’s syndrome T
64. Neuro fibrillary tangles are seen in Pick’s disease F
65. Neuro fibrillary tangles are seen in Lewy body dementia T
66. Neuro fibrillary tangles are seen in Punch drunk syndrome T
67. Parietal lobe lesions can produce superior quadrant hemianopia F
68. Dominant parietal lobe lesions can produce left-right disorientation T
69. Parietal lobe lesions can produce hemi-somatoagnosia T
70. Parietal lobe lesions can produce alexia with agraphia T
71. Non-dominant parietal lobe lesions can produce Sensory dissociation F
72. Non-dominant parietal lobe lesions can lead finger agnosia F
73. Dominant parietal lobe lesions can lead to agraphia T
74. Posterior parietal lobe damage can lead to fluent aphasia. T
75. GCS is a good predictor of long term outcome in severe head injury F
76. Post traumatic epilepsy is more common after a close head injury F
77. The length of post traumatic amnesia correlates with cognitive impairment T
78. Blood is always found in the CSF in severe head injuries F
79. in severe head injuries the brain is more seriously injured when it is in a fixed position F
80. Clouding of consciousness is seen in Korsakoff’s psychosis F
81. Clouding of consciousness is seen in Wernicke’s encephalopathy T
82. Clouding of consciousness is seen in Alcohol hallucinosis F
83. Clouding of consciousness is seen in Gertsman syndrome F
84. Clouding of consciousness is seen in Ganser syndrome T
85. In normal EEG alpha waves are attenuated when eyes are open T
86. In normal EEG alpha waves can have different frequency in 50%of patients T
87. In normal EEG alpha waves may disappear in hepatic coma T
88. In normal EEG alpha waves are seen predominantly in the occipital region T
89. In normal EEG alpha waves are increased by lithium F
90. The caudate is part of limbic system
91. The caudate is altered in Parkinson disease
92. The caudate is connected to substantia nigra
93. The caudate is part of telecephalon
94. The caudate contains glia
95. Oculomotor nerve carry parasympathetic nerve fibres
96. Facial nerve carry parasympathetic nerve fibres
97. Trigeminal nerve carry parasympathetic nerve fibres
98. Vagus nerve carry parasympathetic nerve fibres T
99. Hypoglossal nerve carry parasympathetic nerve fibres F
100. Features of dominant parietal lobe lesions include astreognosis T
101. Features of dominant parietal lobe lesions include dyscalculia T
102. Features of dominant parietal lobe lesions include Sensory inattention T
103. Motor neurone disease may present with Schizophrenoform Psychosis F
104. Motor neurone disease may present with Frontal lobe atrophy or dementia F
105. Motor neurone disease may present with emotional lability T
106. Motor neurone disease may present with spastic paraplegia T
107. More than 2% glucose in CSF is seen in Multiple Sclerosis F
108. More than 2% glucose in CSF Guillane-Barre syndrome T
109. More than 2% glucose in CSF Spinal epidural abscess T
110. More than 2% glucose in CSF Dementia F
111. More than 2% glucose in CSF Acoustic neuroma F
112. Multiple Sclerosis may present with Dysphagia F
113. Multiple Sclerosis may present with Euphoria T
114. Multiple Sclerosis may present with Depression T
115. Multiple Sclerosis may present with Dysarthria T
116. Multiple Sclerosis may present with Cortical blindness
117. Ejaculation is associated with increased parasympathetic activity T
118. Relaxation of the ciliary muscles is associated with increased parasympathetic activity F
119. Relaxation of external anal sphincter is associated with increased parasympathetic activity F
120. Sweating is associated with increased parasympathetic activity T
121. Salivation is associated with increased parasympathetic activity T
122. EEG alpha rhythm decreases to 8Hz after 65 T
123. Temazepam can increase beta fast wave activity on EEG T
124. EEG shows diffuse slow waves even after a month following ECT T
125. Normal EEG makes the diagnosis of Advanced Alzheimer’s disease unlikely T
126. Normal EEG makes the diagnosis of Petit mal epilepsy unlikely F
127. Normal EEG makes the diagnosis of Hypsarrythmia unlikely T
128. Normal EEG makes the diagnosis of sub-acute Sclerosing leuco- encephalitis unlikely T
129. Normal EEG makes the diagnosis of Cerebellar glioma unlikely T
Hypsarrythmia is an age related EEG pattern usually associated with infantile spasm & west syndrome
130. Caudate nucleus is involved in Parkinson’s disease
131. Caudate nucleus together with lenticular nucleus forms the striatum
132. Caudate nucleus projects to sustantia nigra
133. Caudate nucleus is rich in dopamine receptors
134. Mild head injury may cause Intolerance to alcohol F
135. Mild head injury may cause Insomnia T
136. Mild head injury may cause dizziness T
137. Mild head injury may cause Hypersensitivity to sound T
138. Mild head injury may cause Epilepsy F
139. Both Ankle & knee jerks are lost in Fredrick’s ataxia T
140. Both Ankle & knee jerks are lost in Cervical spondylosis F
141. Both Ankle & knee jerks are lost in Polyneuropathy T
142. Both Ankle & knee jerks are lost in Tabes dorsalis F
143. Both Ankle & knee jerks are lost in MND F
144. Eye signs are seen in Tay sachs disease T
145. Eye signs are seen in Phenile keton urea F
146. Eye signs are seen in Wilson’s disease T
147. Eye signs are seen in Wernicke’s encephalopathy T
148. Eye signs are seen in Multiple Sclerosis T
149. Imitative behaviour suggests a frontal lobe lesion F
150. Perseveration suggests a frontal lobe lesion T
151. Having difficulties in card sorting test suggests a frontal lobe lesion T
152. Dyscalculia suggests a frontal lobe lesion
153. Disinhibited behaviour suggests a frontal lobe lesion
154. Sequelae of closed head injury include Insomnia T
155. Sequelae of closed head injury include Epilepsy T
156. Sequelae of closed head injury include Intolerance to Alcohol F
157. Sequelae of closed head injury include Hyper-sensitivity to sound T
158. Sequelae of closed head injury include memory loss T
Retro grade amnesia (time between the injury & last memory of events before injury) is a Poor prognostic indicator.
Anterograde amnesia (time between injury & recovery of normal memory) is a good prognostic indicator.
The longer the amnesia the worse the prognosis. Intra cranial bleeding & penetrating injuries are associated with poor prognosis.
Sequelae of head injury include:
· Neurosis, depression, fatigue, anxiety, irritability, excessive sensitivity to noise & somatic complaints.
· Personality change
· Psychosis
· Cognitive impairment
· Epilepsy especially with penetrating injuries
159. Neurological examination in Parkinson’s disease reveals large sweeping hand writing F
160. Neurological examination in Parkinson’s disease reveals decrease voluntary muscle power F
161. Neurological examination in Parkinson’s disease reveals Increased fatigability F
162. Neurological examination in Parkinson’s disease reveals Bradykinesia T
163. Neurological examination in Parkinson’s disease reveals Failed habituation to glabellar tab T
164. Characteristic features of CJD include association with a transmissible agent T
165. Characteristic features of CJD include Myoclonic jerks T
166. Characteristic features of CJD include Ataxia T
167. Characteristic features of CJD include spontaneous remission F
168. Characteristic features of CJD include akinetic mutism in late stages T
169. Characteristic features of CJD include repetitive sharp wave complexes on EEG T
170. Frontal lobe meningioma may cause calcification on skull X ray T
171. Frontal lobe meningioma may cause epilepsy T
172. Frontal lobe meningioma may cause ataxia T
173. Frontal lobe meningioma may cause personality change T
174. Frontal lobe meningioma is best diagnosed by CT scan F
175. Features of left parietal lobe lesion include apathy & psychomotor retardation F
176. Features of left parietal lobe lesion include alexia & agraphia T
177. Features of left parietal lobe lesion include hemi-somatoagnosia F
178. Features of left parietal lobe lesion include amnesia F
179. Features of left parietal lobe lesion include Euphoria F
180. Fast beta activity on EEG may be caused by Chlorpromazine F
181. Fast beta activity on EEG may be caused by Amitryptiline F
182. Fast beta activity on EEG may be caused by phenobarbiton T
183. Fast beta activity on EEG may be caused by Lithium F
184. Fast beta activity on EEG may be caused by Diazepam T
185. De javu is one of the paramnesias T
186. De javu can be part or a whole of the aura in temporal lobe epilepsy T
187. De javu is a spurious sense of unfamiliarity F
188. De javu is not compelling as a feeling & is never remembered F
189. De javu occurs in normal people T
190. Signs in Huntington’s chorea present characteristically in late teens F
191. Huntington’s chorea is autosomal dominant T
192. Huntington’s chorea may display anticipation through generations T
193. In Huntington’s chorea psychotic features may precede abnormal movements T
194. Atrophy of the caudate nucleus is a typical feature of Huntington’s chorea T
195. Personality disorder is more common in idiopathic than epilepsy of known aetiology F
196. Personality disorder is commoner in grand mal than in petit mal T
197. There is a specific epileptic personality F
198. Hyper sexuality is the commonest sexual disorder in epilepsy F
199. Primary epilepsy account for <50% of all patients F
200. Retrograde amnesia is a valid & valuable guide to the severity of head injury F
201. Pre morbid personality is important in determining ensuing psychiatric disturbance in patient with head injury T
202. Post-traumatic epilepsy develops in 25% of patients following head injury
203. Cognitive impairment is the commonest psychiatric disorder following head injury F
204. The incidence of psychiatric Sequelae is higher in children following head injury F
205. A parietal lobe lesion may cause motor Jacksonian fits F
206. A parietal lobe lesion may cause astereognosis T
207. A parietal lobe lesion may cause grasp reflex F
208. A parietal lobe lesion may cause anosognosia T
209. A parietal lobe lesion may cause homonymous hemianopia T
210. Frontal lobe syndrome is frequently associated with hyper-sexuality F
211. Frontal lobe syndrome is frequently associated with fatuous mood T
212. Frontal lobe syndrome is frequently associated with disinhibition T
213. Frontal lobe syndrome is frequently associated with behavioural changes in the absence of neurological signs T
214. Frontal lobe syndrome is frequently associated with marked loss of memory F
215. Multiple sclerosis is associated with Optic neuritis T
216. Multiple sclerosis is associated with Euphoria T
217. Multiple sclerosis is associated with nystagmus T
218. Abnormalities of gait are commonly found in normal pressure hydrocephalus T
219. Abnormalities of gait are commonly found in Folate deficiency F
220. Abnormalities of gait are commonly found in Fredrick’s ataxia T
221. Abnormalities of gait are commonly found in Sturge weber syndrome F
222. Abnormalities of gait are commonly found in Parkinson’s disease T
223. Cerebellar lesions may cause Hypotonicity T
224. Cerebellar lesions may cause reduced deep tendon reflexes T
225. Cerebellar lesions may cause tremor at rest F
226. Cerebellar lesions may cause Dysarthria T
227. Cerebellar lesions may cause sphincter disturbance F
228. Loss of sensation is seen immediately following acute transection of the spinal cord T
229. Loss of muscle power is seen immediately following acute transection of the spinal cord T
230. Spasticity is seen immediately following acute transection of the spinal cord F
231. Loss of reflexes is seen immediately following acute transection of the spinal cord T
232. Vasodilatation is seen immediately following acute transection of the spinal cord T
233. Frontal lobe lesions are associated with inability to plan actions T
234. Frontal lobe lesions are associated with apathy T
235. Frontal lobe lesions are associated with Positive grasp reflex T
236. Frontal lobe lesions are associated with incontinence T
237. Frontal lobe lesions are associated with aphasia T
238. Frontal lobe lesions are associated with contra-lateral upper limb weakness T
239. Frontal lobe lesions are associated with topographical disorientation F
240. Parietal lobe lesions are associated with topographical disorientation T
241. Parietal lobe lesions are associated with Visual agnosia
242. Parietal lobe lesions are associated with Sensory inattention T
243. Parietal lobe lesions are associated with Urinary incontinence F
244. Parietal lobe lesions are associated with De Javu phenomenon F
245. Asymmetrical neurological signs are characteristic of idiopathic Parkinson’s F
246. Action tremor is characteristic of idiopathic Parkinson’s F
247. Spastic rigidity is characteristic of idiopathic Parkinson’s F
248. Brady kinesia is characteristic of idiopathic Parkinson’s T
249. Difficulty turning in bed is characteristic of idiopathic Parkinson’s T
250. Paralysis of the right leg is seen after anterior coronary artery occlusion T
251. Paralysis of the left arm is seen after anterior coronary artery occlusion F
252. Grasp reflex is seen after anterior coronary artery occlusion T
253. Right homonymous hemianopia is seen after anterior coronary artery occlusion F
254. Bruca’s aphasia is seen after anterior coronary artery occlusion F
255. Communicating hydrocephalus can cause raised intra-cranial pressure F
256. Sub-arachnoid haemorrhage can cause raised intra-cranial pressure T
257. Sagital vein thrombosis can cause raised intra-cranial pressure T
258. Sub acute sclerosing panenceohalitis can cause raised intra-cranial pressure F
259. Hypertensive encephalopathy can cause raised intra-cranial pressure T
260. Echopraxia is a feature of frontal lobe syndrome F
261. Characteristic features of non dominant parietal lobe lesion include inability to appreciate music F
262. Characteristic features of non dominant parietal lobe lesion inability to plan ahead F
263. Characteristic features of non dominant parietal lobe lesion inability to draw from memory T
264. Characteristic features of non dominant parietal lobe lesion inability to shift from one concept to another F
265. Characteristic features of non dominant parietal lobe lesion inability to learn new words F
266. Characteristic features of communicating hydrocephalus include onset in the second decade of life
267. Characteristic features of communicating hydrocephalus include Osteoporotic changes detected on skull X ray
268. Characteristic features of communicating hydrocephalus include disturbance of gait T
269. Characteristic features of communicating hydrocephalus include dementia T
270. Characteristic features of communicating hydrocephalus include pathognomonic changes on EEG
271. Posterior column of the spinal cord carry sensory impulses concerned with Pain F
272. Posterior column of the spinal cord carry sensory impulses concerned with proprioception T
273. Posterior column of the spinal cord carry sensory impulses concerned with light touch T
274. Posterior column of the spinal cord carry sensory impulses concerned with hot & cold sensation F
275. Posterior column of the spinal cord carry sensory impulses concerned with Vibration sensation T
276. Nystagmus is a recognised feature of Vertibro-basillar artery insufficiency T
277. Nystagmus is a recognised feature of multiple sclerosis T
278. Nystagmus is a recognised feature of Horner’s syndrome F
279. Nystagmus is a recognised feature of barbiturate abuse T
280. Nystagmus is a recognised feature of cerebello-pontine angle tumours T
281. Occlusion of middle cerebral artery leads to partial epsilateral facial weakness F
282. Occlusion of middle cerebral artery leads to contra-lateral exaggerated deep reflexes T
283. Occlusion of middle cerebral artery leads to epsilateral spastic paralysis F
284. Occlusion of middle cerebral artery leads to absence of plantar reflexes F
285. Occlusion of middle cerebral artery leads to difficulties in understanding both spoken & written words T
286. Amnestic syndrome may be caused by bilateral hypocampal infarction T
287. Amnestic syndrome may be caused by Epilepsy T
288. Amnestic syndrome may be caused by Herpes simplex encephalitis T
289. Amnestic syndrome may be caused by Infarction of the sub-thalamic body F
290. Amnestic syndrome may be caused by Closed head injury T
291. Amnestic syndrome may be caused by chronic alcohol abuse T
292. Constructional apraxia frequently appear with disorders of spatial perception T
293. In head injuries post traumatic amnesia can occur without retrograde amnesia T
294. TLE is associated with hypo-sexuality T
295. The juvenile onset Huntington’s chorea has a higher incidence of psychosis than adult onset type T
296. Slowing of speech is an early sign of AIDS dementia T
297. Inappropriate tearfulness is a feature of multiple sclerosis T
298. The duration of retrograde amnesia is useful in determining prognosis after head injury F
299. CT scan is superior to MRI In detecting calcified brain lesions T
300. Automatism is usually seen in clear consciousness F
301. Athetotic movements disappear in sleep T
302. Tinnitus is a characteristic feature of TLE F
303. Jamais vu is a characteristic feature of TLE T
304. Epigastric distress is a characteristic feature of TLE T
305. Olfactory hallucination is a characteristic feature of TLE T
306. Errors of judgement differentiate FL syndrome from mania F
307. Urinary incontinence is a characteristic feature of TLE F
308. Speech centres are normally located in the left cerebral hemispheres T
309. Speech production of the average 2-year-old is known as telegraphic speech T
310. Lumber puncture should be avoided in patients suspected clinically of having cerebral abscess T
311. In Huntington’s chorea rhythmical high amplitude delta activity on the EEG is characteristic F
312. Lesions of the ventro-medial hypothalamus cause increased appetite T
313. Men are more commonly affected by Huntington disease than women F
314. Cognitive impairment occurs early in the course of Huntington disease F
315. Violence is rare during automatism T
316. In De javu the person has feeling of familiarity for events experienced for the first time T
317. In jamais vu an experience that a person has experienced before is not associated with the appropriate feeling of familiarity T
318. The most common psychiatric disorder seen in MS is euphoria F
319. Logoclonia may occur in Parkinson’s T
320. Naming & reading are rarely impaired in pure word deafness F
321. Speech is fluent in jargon Dysphasia T
322. In alexia with agraphia the patient is unable to read, write, speak or understand speech F
323. In Huntington disease the insight is lost early in the course of illness T
324. Tics occur at an equal rate in males & females F F>M
325. Behaviour during automatism is usually inappropriate & purposeless F
326. Olfactory hallucination in epilepsy strongly suggest the presence of a brain tumour F
327. The right side of the brain is the dominant side in most left handed people F
328. In pure word dumbness writing is preserved but speech can not be produced at will T
329. Lesions in the bruca’s area produce expressive dysphasia T
330. Lesion of the dominant parietal lobe causes visuo-spatial problems T
331. Post stroke depression is more common following left sided stroke
332. About 50% of people with learning disabilities suffer from epilepsy
333. Bulbar & pseudo-bulbar palsy can result in logoclonia F
334. In conduction dysphasia the patient can not speak & write but can repeat whatever is spoke to him F
335. In pure agraphia speech is normal T
336. Hemisomato-agnosia is the neglect of one side of the body T
337. Somatop-agnosia is an inability to recognise a neurological deficit occurring in one self F
